TY - JOUR
T1 - Sominone improves memory impairments and increases axonal density in Alzheimer's disease model mice, 5XFAD
AU - Joyashiki, Eri
AU - Matsuya, Yuji
AU - Tohda, Chihiro
N1 - Funding Information:
This work was partially supported by a grant for Expansion Program in the Knowledge Cluster Initiative from the Ministry of Education, Culture, Sports, Science and Technology, Japan, and the Takeda Science Foundation. Address correspondence to Assistant Professor Chihiro Tohda, Ph.D., Division of Biofunctional Evaluation, Research Center for Ethnomedicine, Institute of Natural Medicine, University of Toyama, Toyama 930-0194, Japan. E-mail: chihiro@inm.u-toyama.ac.jp
PY - 2011/4
Y1 - 2011/4
N2 - Previously we showed that steroidal sapogenin, sominone improved memory after a single i.p. injection into normal mice. However, it had not been reported that sominone could recover memory deficits in a severe Alzheimer's disease (AD) model animal. Therefore, we aimed to investigate that sominone improved memory impairments in the 5XFAD mouse, model for AD. In the current study, we used sominone that we had synthesized. 5XFAD mice were given 10 μmol/kg sominone intraperitoneally for 9 days. In addition to object recognition memory, axonal density, amyloid plaque number, and activated microglia in the brain were evaluated. Sominone treatment significantly improved object recognition memory compared with vehicle control treatment. Sominone treatment significantly enhanced axonal densities in the frontal cortex and parietal cortex but had no effects on amyloid plaque number and activated microglia. In cultured cortical neurons, the axonal length was significantly reduced by Aβ(1-42) treatment. However, that was markedly recovered 5 days after the treatment with 1 μM sominone. Neuronal loss was not observed in the cortex and hippocampus of 5XFAD mice at 6-8 months of age. These results suggest that memory deficits in AD may be improved by sominone independently of reducing amyloid plaques and neuroinflammation.
AB - Previously we showed that steroidal sapogenin, sominone improved memory after a single i.p. injection into normal mice. However, it had not been reported that sominone could recover memory deficits in a severe Alzheimer's disease (AD) model animal. Therefore, we aimed to investigate that sominone improved memory impairments in the 5XFAD mouse, model for AD. In the current study, we used sominone that we had synthesized. 5XFAD mice were given 10 μmol/kg sominone intraperitoneally for 9 days. In addition to object recognition memory, axonal density, amyloid plaque number, and activated microglia in the brain were evaluated. Sominone treatment significantly improved object recognition memory compared with vehicle control treatment. Sominone treatment significantly enhanced axonal densities in the frontal cortex and parietal cortex but had no effects on amyloid plaque number and activated microglia. In cultured cortical neurons, the axonal length was significantly reduced by Aβ(1-42) treatment. However, that was markedly recovered 5 days after the treatment with 1 μM sominone. Neuronal loss was not observed in the cortex and hippocampus of 5XFAD mice at 6-8 months of age. These results suggest that memory deficits in AD may be improved by sominone independently of reducing amyloid plaques and neuroinflammation.
KW - 5XFAD
KW - Alzheimer's disease
KW - axon
KW - object recognition memory
KW - sominone
UR - http://www.scopus.com/inward/record.url?scp=79951883328&partnerID=8YFLogxK
U2 - 10.3109/00207454.2010.541571
DO - 10.3109/00207454.2010.541571
M3 - 学術論文
C2 - 21329473
AN - SCOPUS:79951883328
SN - 0020-7454
VL - 121
SP - 181
EP - 190
JO - International Journal of Neuroscience
JF - International Journal of Neuroscience
IS - 4
ER -