Helicobacter pylori Induces Serine Phosphorylation of EGFR via Novel TAK1-p38 Activation Pathway in an HB-EGF-Independent Manner

Keyphrases

• Activation Mechanism 100%
• Helicobacter Pylori (H. pylori) 100%
• P38 Activation 100%
• HB-EGF 100%
• Serine Phosphorylation 100%
• Phosphorylation 50%
• Infected Cells 30%
• Flow Cytometry 20%
• NF-B 20%
• Small Interfering RNA (siRNA) 20%
• Hepatitis B Surface Antigen (HBsAg) 20%
• Serine Residues 20%
• CagA 20%
• Transcription Factor 10%
• Transcriptional Activation 10%
• Western Blotting 10%
• RNA Interference 10%
• MKN-45 Cells 10%
• Gefitinib 10%
• P38 MAPK Pathway 10%
• Gastric Epithelial Cells 10%
• Fluorescence Microscopy 10%
• Immunofluorescence 10%
• EGFR Phosphorylation 10%
• Chemical Inhibitors 10%
• Gastric Carcinoma 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Phosphotransferase 100%
• Epidermal Growth Factor Receptor 100%
• Serine 100%
• Helicobacter Pylori 100%
• Heparin Binding Epidermal Growth Factor 100%
• Flow Cytometry 18%
• Small Interfering RNA 18%
• Hepatitis B Antibody 18%
• Transcription Factors 9%
• Western Blot 9%
• Gefitinib 9%
• Carcinogenesis 9%
• Fluorescence Microscopy 9%
• CagA 9%

Immunology and Microbiology

• Helicobacter Pylori 100%
• Pylorus 100%
• Heparin Binding Epidermal Growth Factor 100%
• Internalization 42%
• Wild Type 28%
• Hepatitis B Surface Antibody 28%
• Immunology 28%
• Western Blot 14%
• Immunofluorescence 14%
• RNA Interference 14%
• Transcription Factors 14%
• CagA 14%