@article{3947a8c032164d56a8721bd1baf5c141,
title = "NGL-2 Deletion Leads to Autistic-like Behaviors Responsive to NMDAR Modulation",
abstract = "NGL-2 is a postsynaptic adhesion molecule known to regulate synaptic transmission, but whether NGL-2 regulates synaptic plasticity and specific behaviors remains unknown. Um et al. find that mice lacking NGL-2 display suppressed NMDA receptor-dependent synaptic plasticity and autistic-like social deficits and repetitive behaviors that are responsive to NMDA receptor activation.",
keywords = "NMDA receptors, autism, repetitive behavior, social interaction, synaptic adhesion",
author = "Um, {Seung Min} and Seungmin Ha and Hyejin Lee and Jihye Kim and Kyungdeok Kim and Wangyong Shin and Cho, {Yi Sul} and Roh, {Junyeop Daniel} and Jaeseung Kang and Taesun Yoo and Noh, {Young Woo} and Yeonsoo Choi and Bae, {Yong Chul} and Eunjoon Kim",
note = "Funding Information: We thank Sungsoo Kim at KAIST for helping with Golgi staining and Eunsil Cho at KIST (Korea Institute of Science and Technology) for helping with the graphical abstract design. This work was supported by the National Research Foundation of Korea (NRF) grant funded by the Korean government (NRF-2017R1A5A2015391 to Y.C.B.) and the Institute for Basic Science (IBS-R002-D1 to E.K.). Funding Information: We thank Sungsoo Kim at KAIST for helping with Golgi staining and Eunsil Cho at KIST (Korea Institute of Science and Technology) for helping with the graphical abstract design. This work was supported by the National Research Foundation of Korea (NRF) grant funded by the Korean government ( NRF-2017R1A5A2015391 to Y.C.B.) and the Institute for Basic Science ( IBS-R002-D1 to E.K.). ",
year = "2018",
month = jun,
day = "26",
doi = "10.1016/j.celrep.2018.05.087",
language = "英語",
volume = "23",
pages = "3839--3851",
journal = "Cell Reports",
issn = "2211-1247",
publisher = "Elsevier BV",
number = "13",
}