Pathophysiological roles of stress-activated protein kinases in pulmonary fibrosis

Keyphrases

• Stress-activated Protein Kinase 100%
• Pulmonary Fibrosis 100%
• Pathophysiological Roles 100%
• P38 Mitogen-activated Protein Kinase (p38 MAPK) 33%
• Epithelium 16%
• Poor Prognosis 16%
• Blood Vessels 16%
• Fibroblasts 16%
• Molecular Mechanism 16%
• Protein Kinase 16%
• Therapeutic Potential 16%
• Connective Tissue 16%
• New Therapeutics 16%
• Macrophages 16%
• Lung 16%
• Fibrogenesis 16%
• Transcriptome Analysis 16%
• Bleomycin-induced Pulmonary Fibrosis 16%
• Cellular Processes 16%
• Alveolar Epithelial Type II Cells 16%
• Fibrotic Lung Disease 16%
• Idiopathic Pulmonary Fibrosis 16%
• RNA-sequencing Data 16%
• Molecular Function 16%
• Lung Cells 16%
• Process Function 16%
• Hematopoietic Tissue 16%
• P38 Signaling Pathway 16%
• C-Jun N-terminal Kinase 16%
• Environmental Stress 16%
• Lymphoid Tissue 16%

Medicine and Dentistry

• Stress Activated Protein Kinase 100%
• Pulmonary Fibrosis 100%
• Mitogen Activated Protein Kinase P38 33%
• Lung 33%
• Epithelial Cell 16%
• Connective Tissue 16%
• Fibrosis 16%
• Macrophage 16%
• Idiopathic Pulmonary Fibrosis 16%
• Gene Expression Profiling 16%
• Protein Kinase C 16%
• Amino Terminal Sequence 16%
• Fibrogenesis 16%
• RNA Sequence 16%
• Hematopoietic Tissue 16%
• Phosphotransferase 16%
• Bleomycin 16%
• Lymphatic Tissue 16%
• Silo-Filler's Disease 16%
• Fibroblast 16%

Pharmacology, Toxicology and Pharmaceutical Science

• Stress Activated Protein Kinase 100%
• Lung Fibrosis 100%
• Mitogen Activated Protein Kinase P38 33%
• Phosphotransferase 16%
• Fibrosis 16%
• Fibrosing Alveolitis 16%
• Protein Kinase C 16%
• Lung Disease 16%
• Bleomycin 16%
• RNA-Sequencing 16%
• Cellular Processes 16%
• Transcriptome 16%