Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

Neil E. Bowles; Cammon B. Arrington; Keiichi Hirono; Tsuneyuki Nakamura; Long Ngo; Yin Shen Wee; Fukiko Ichida; John H. Weis

doi:10.1002/mgg3.79

Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

Neil E. Bowles^*, Cammon B. Arrington, Keiichi Hirono, Tsuneyuki Nakamura, Long Ngo, Yin Shen Wee, Fukiko Ichida, John H. Weis

^*この論文の責任著者

糸魚川・地域医療支援学講座

研究成果: ジャーナルへの寄稿 › Letter › 査読

3 被引用数 (Scopus)

本文言語	英語
ページ（範囲）	356-361
ページ数	6
ジャーナル	Molecular Genetics and Genomic Medicine
巻	2
号	4
DOI	https://doi.org/10.1002/mgg3.79
出版ステータス	出版済み - 2014/07

ASJC Scopus 主題領域

分子生物学
遺伝学
遺伝学（臨床）

文献へのアクセス

10.1002/mgg3.79

引用スタイル

Bowles, N. E., Arrington, C. B., Hirono, K., Nakamura, T., Ngo, L., Wee, Y. S., Ichida, F., & Weis, J. H. (2014). Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions. Molecular Genetics and Genomic Medicine, 2(4), 356-361. https://doi.org/10.1002/mgg3.79

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title = "Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions",

author = "Bowles, {Neil E.} and Arrington, {Cammon B.} and Keiichi Hirono and Tsuneyuki Nakamura and Long Ngo and Wee, {Yin Shen} and Fukiko Ichida and Weis, {John H.}",

year = "2014",

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doi = "10.1002/mgg3.79",

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Bowles, NE, Arrington, CB, Hirono, K, Nakamura, T, Ngo, L, Wee, YS, Ichida, F & Weis, JH 2014, 'Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions', Molecular Genetics and Genomic Medicine, vol. 2, no. 4, pp. 356-361. https://doi.org/10.1002/mgg3.79

Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions. / Bowles, Neil E.; Arrington, Cammon B.; Hirono, Keiichi その他.
In: Molecular Genetics and Genomic Medicine, Vol. 2, No. 4, 07.2014, p. 356-361.

研究成果: ジャーナルへの寄稿 › Letter › 査読

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T1 - Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

AU - Bowles, Neil E.

AU - Arrington, Cammon B.

AU - Hirono, Keiichi

AU - Nakamura, Tsuneyuki

AU - Ngo, Long

AU - Wee, Yin Shen

AU - Ichida, Fukiko

AU - Weis, John H.

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M3 - Letter

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JO - Molecular Genetics and Genomic Medicine

JF - Molecular Genetics and Genomic Medicine

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